Anca Mitroi

Scopus Publications

Diagnostic Value of miR-21, miR-122, miR-145, and miR-146a in Acute Pancreatitis: Findings from the PASEVO Cohort in Southeastern Romania
Diana Iosif, Costel Stelian Brînzan, Andra Iulia Suceveanu, Iulia Cîndea, Viorel Gherghina, et al.
International Journal of Molecular Sciences, 2026
Acute pancreatitis (AP) is an inflammatory condition of the pancreas that initiates a cascade of inflammation, which can cause pancreatic damage or spread to other organs. The severity of AP ranges from mild acute pancreatitis (MAP) to severe acute pancreatitis (SAP). While standard diagnostic tools include contrast-enhanced CT scans, clinical scoring systems like APACHE II and Ranson, and laboratory tests, miRNAs have emerged as crucial regulators of gene expression involved in various physiological and pathological processes. Over the last decade, research has demonstrated that significant alterations in miRNA levels are integral to AP development. Since one miRNA can target multiple genes, they serve as key modulators of disease progression.
Interplay Between Immune Microenvironment CD8+ Tumor-Infiltrating Lymphocytes and PDL-1 Expression as Prognostic Markers in Invasive Cervical Squamous Cell Carcinoma
Laura-Andra Petrică, Mariana Deacu, Georgeta Camelia Cozaru, Anca Florentina Mitroi, Gabriela Izabela Bălţătescu, et al.
Medicina Lithuania, 2025
Background: Cervical cancer remains a major cause of cancer-related morbidity and mortality worldwide, with limited therapeutic options for advanced disease. As we better understand the fine mechanisms involved in the interaction between tumor cells and the tumor microenvironment, new paths and opportunities will emerge. Recent evidence highlights the prognostic and predictive roles of immune checkpoint markers and tumor-infiltrating lymphocytes (TILs), especially CD8+ TILs, in shaping treatment outcomes. Objectives: This study investigated the immunohistochemical expression of PD-L1 and CD8+ TILs in 48 newly diagnosed, treatment-naive cervical cancer cases and analyzed their associations with clinicopathological features and survival outcomes. Results: In our cohort, we observed that PD-L1 positivity was identified in 68.8% of cases, most frequently in advanced FIGO stages and in tumors with lympho-vascular invasion or with a high proliferation rate evaluated by the Ki-67 index. High levels of intra-tumoral CD8+ TILs were observed in 52.1% of cases and correlated positively with stromal TILs, lower proliferation rates, and absence of vascular invasion. A significant inverse relationship was found between PD-L1 expression and the density of CD8+ TILs (p = 0.047). Survival analysis showed that patients exhibiting a “cold” immunophenotype with low levels of CD8+ TILs and PD-L1-positive tumors had worse outcomes, while high levels of CD8+ TILs played a protective role. Conclusions: Our study highlights the importance of the immunohistochemical assessment of PD-L1 and CD8+ TILs biomarkers, which have a complementary inter-relationship and have a significant prognostic impact on cervical squamous cell carcinoma. PD-L1 positivity marks aggressive disease features, while higher intra-tumoral CD8+ TIL density is protective. Their combined evaluation may improve patient stratification and inform immunotherapy strategies.
The Impact of Polymorphisms on Complex Medical Rehabilitation Treatment in Patients with Sarcopenia and Sarcopenic Obesity (SARCOGEN)
Andreea-Dalila Nedelcu, Liliana-Elena Stanciu, Anca-Florentina Mitroi, Lucian-Cristian Petcu, Carmen Oprea, et al.
Balneo and Prm Research Journal, 2025
Sarcopenia and sarcopenic obesity are conditions that are underdiagnosed in medical practice. Given their significant impact on quality of life with advancing age, identifying these disorders and their underlying causes, as well as developing targeted intervention strategies, are major areas of interest in current medical research. Investigating single nucleotide polymorphisms (SNPs) associated with these conditions, particularly in patients admitted to rehabilitation units, may open new avenues for diagnosis, prevention and treatment. This study conducts a rigorous review of the literature to evaluate the relationship between polymorphisms, sarcopenia and sarcopenic obesity, while also examining how the response to rehabilitation treatment may vary. Three polymorphisms were studied as having a significant influence on sarcopenia and sarcopenic obesity through their involvement in specific functional pathways. Among these, one polymorphism has been extensively studied in relation to medical rehabilitation, with notable associations observed between genotype and treatment response. Implementing screening for sarcopenia and sarcopenic obesity, along with genetic polymorphism analysis, represents a promising research direction. The findings of this preliminary study make a meaningful contribution to the current medical literature and may support the development of personalized rehabilitation interventions for affected patients.
The miR-21-5p, miR-30c-5p, and miR-182-5p as Biomarkers in Clear Cell Renal Cell Carcinoma: A Southeastern Romanian Cohort Study
Ionuț Burlacu, Mariana Așchie, Georgeta Camelia Cozaru, Mariana Deacu, Gabriela Miruna Vizireanu, et al.
Genes, 2025
Background: This study aimed to evaluate the expression and clinical relevance of three mature miRNAs (miR-21-5p, miR-30c-5p, and miR-182-5p) in patients with clear cell renal cell carcinoma (ccRCC) from southeast Romania, and to explore their potential as non-invasive diagnostic and prognostic biomarkers. Methods: miRNA expression levels were measured using TaqMan® MGB and qRT-PCR in paired tumor and adjacent non-cancerous tissues, as well as in serum-derived exosomes, from 26 ccRCC patients. Statistical analysis included the Wilcoxon test for group comparisons and non-parametric tests for correlations with clinicopathological features. Receiver operating characteristic (ROC) curves were used to assess diagnostic performance, and miRNA panels were constructed for improved accuracy. Results: Significant dysregulation of the investigated miRNAs was observed. miR-21-5p was markedly overexpressed in both tumor tissues (3.46-fold, p < 0.001) and serum exosomes (3.26-fold, p < 0.001). miR-182-5p showed modest overexpression in tissues (0.56-fold, p < 0.001) and serum (0.85-fold, p < 0.001), whereas miR-30c-5p was significantly downregulated in both tissues (2.48-fold decrease, p < 0.001) and serum exosomes (2.29-fold decrease, p = 0.0003). Elevated miR-182-5p expression correlated with tumor localization in the right kidney (p = 0.02) and lymph node involvement (p = 0.04). Similarly, higher miR-21-5p levels in serum exosomes were associated with right-sided tumors (p = 0.01). ROC analysis revealed distinct expression profiles for all three miRNAs between ccRCC and normal tissue, both in tissue and exosomal samples (all p < 0.05). Combined biomarker panels yielded high diagnostic performance (AUC = 0.94 for tissue, AUC = 0.93 for exosomes). Conclusions: This study underscores the potential of miR-21-5p, miR-30c-5p, and miR-182-5p as non-invasive biomarkers for ccRCC diagnosis and prognosis. The use of serum exosomal miRNA panels offers a promising alternative to tissue-based diagnostics in Romanian ccRCC patients.
Toward Personalized Surgery in Advanced Prostate Cancer: Stratification by PTEN, AR-V7, TP53, TMPRSS2-ERG, and ERBB2 Genetic Alterations
Cristina Anita Ionescu (Mitu), Georgeta Camelia Cozaru, Mariana Aschie, Nicoleta Leopa, Bogdan Cimpineanu, et al.
Chirurgia Romania, 2025
Background: Advanced prostate cancer is a biologically heterogeneous disease often marked by multiple genetic and epigenetic alterations that influence tumor progression, treatment resistance, and prognosis. Among the most frequently altered genes are PTEN, AR-V7, TP53, TMPRSS2-ERG, and ERBB2, each with potential relevance for stratifying risk and guiding targeted therapy. Methods: This retrospective study included 43 patients with advanced prostate cancer who underwent radical prostatectomy. Tumor specimens were analyzed using fluorescence in situ hybridization (FISH) to assess the mutational status of the five markers. Clinicopathological parameters, including PSA levels, Gleason score, tumor stage, and invasion status, were correlated with molecular alterations using multinomial logistic regression. Results: The most common isolated alteration was PTEN loss (20.9%), followed by TP53 amplification (16.3%), TMPRSS2-ERG fusion (13.9%), AR-V7 expression (11.6%), and ERBB2 amplification (7%). Combined alterations were also observed, with dual or triple marker expression in select aggressive cases. PTEN- and AR-V7+ were associated with low PSA values despite aggressive pathology, while ERBB2+ correlated with high PSA levels and high Gleason scores. TP53+ and ERBB2+ were also significantly associated with high-grade tumors (Gleason 7). AR-V7+ was the only marker significantly associated with seminal vesicle invasion. Younger age was weakly correlated with AR-V7+ and TP53+ status. Conclusions: The molecular profile defined by PTEN, AR-V7, TP53, and ERBB2 identifies distinct biological subtypes in advanced prostate cancer, each with specific prognostic and therapeutic implications. Integration of these biomarkers into routine clinical assessment may improve treatment personalization and risk stratification. Validation in larger, prospective cohorts is warranted.
Comparative Clinical-Imaging and Histogenetic Analysis Between Astrocytoma IDH-Mutant Grade 4 and Glioblastoma IDH-Wildtype—Is There Really a Worse One?
Cristian Ionut Orasanu, Mariana Aschie, Mariana Deacu, Madalina Bosoteanu, Sorin Vamesu, et al.
Diagnostics, 2025
Background: Brain tumors pose a significant health threat, leading to high morbidity and mortality rates. Astrocytoma IDH-mutant grade 4 (A4IDHmt) and glioblastoma IDH-wildtype (G4IDHwt) exhibit similar clinical and imaging characteristics. This study aims to highlight the differences in their clinical evolution and histogenetic aspects with the possible therapeutic impact, as well as the adverse prognostic factors in patient survival. Methods: We performed a 10-year retrospective study of grade 4 gliomas, evaluating immunomarkers and FISH tests. We also quantified tumor necrosis and microvascular density. Results: A total of 81 cases were identified; 54.32% were A4IDHmt. We observed that A4IDHmt patients were younger (34.10% under 50) and had a higher survival rate (4.55%). This group also exhibited a more pronounced microvascular density (p = 0.010) and proliferative index (p = 0.026). G4IDHwt was associated with larger tumor volumes (94.84 cm3 vs. 86.14 cm3), lower resectability rates (82.88% vs. 87.67%), and a more significant immature cell population (83.78% vs. 68.18%). In the case of both, the negative risk on survival in the univariate analysis is given by advanced age (A4IDHmt: HR = 1.035, G4IDHwt: HR = 1.045) and p53 immunopositivity (A4IDHmt: HR = 6.962, G4IDHwt: HR = 4.680). Conclusions: The negative risk factors for A4IDHmt include the rapid onset of clinical symptoms (HR = 2.038), diabetes mellitus (HR = 2.311), arterial hypertension (HR = 2.325), residual tumor (HR = 2.662), increased residual tumor volume (HR = 1.060), increased microvascular density (HR = 1.096), and high tumor necrosis (HR = 1.097). For G4IDHwt, the negative risk factors consist of increased residual volume (HR = 1.023), lost PTEN immunoreaction (HR = 33.133), and unmethylated DNA status (HR = 6.765, respectively HR = 20.573). Even if it has more risk factors, A4IDHmt is the lesser evil.
Is It Possible to Prevent the Thanatogenetic Processes in Premature Babies?
Sinziana Andra Ghitoi, Mariana Deacu, Mariana Aschie, Manuela Enciu, Anca Florentina Mitroi, et al.
Clinics and Practice, 2024
Preterm births comprise all pregnancies coming to an end before the gestational age of 37 weeks and remain the leading cause of death in children under 5 years old despite efforts to reduce their occurrence. We aim to analyze all morbidity and mortality data to understand causes and risk factors, helping in prevention efforts. This study includes 140 cases collected during 2018–2022. Demographic, maternal, and thanatogenetic data were statistically analyzed. We observed an upward slope of stillborn babies. In the case of live-born premature, the average survival was 301.76 h. The multivariate analysis noted that extremely low birth weight (HR = 5.141) and very low birth weight (HR = 4.177) are risk factors involved in mortality. Increased parity was associated with premature births with low and very low birth weight (p = 0.019). We observed that a mother’s age of over 30 years is predictable for the development of pregnancy-induced hypertension. Cerebral and pulmonary hemorrhages were the most common intermediate morbid conditions, with prematurity and plurivisceral hemorrhages serving as their root causes. We have identified that anthropometric measurements have a high predictability on malformed babies. The identified associations indicate a shared mechanism for certain lesion processes, which can help optimize resources for predicting and preventing preterm neonatal issues.
Cell death and DNA damage via ROS mechanisms after applied antibiotics and antioxidants doses in prostate hyperplasia primary cell cultures
Elena Matei, Anita Cristina Ionescu, Manuela Enciu, Violeta Popovici, Anca Florentina Mitroi, et al.
Medicine United States, 2024
Tumor heterogeneity results in aggressive cancer phenotypes with acquired resistance. However, combining chemical treatment with adjuvant therapies that cause cellular structure and function perturbations may diminish the ability of cancer cells to resist at chemical treatment and lead to a less aggressive cancer phenotype. Applied treatments on prostate hyperplasia primary cell cultures exerted their antitumor activities through mechanisms including cell cycle blockage, oxidative stress, and cell death induction by flow cytometry methods. A 5.37 mM Chloramphenicol dose acts on prostate hyperplasia cells by increasing the pro-oxidant status, inducing apoptosis, autophagy, and DNA damage, but without ROS changes. Adding 6.30 mM vitamin C or 622 µM vitamin E as a supplement to 859.33 µM Chloramphenicol dose in prostate hyperplasia cells determines a significant increase of ROS level for a part of cells. However, other cells remain refractory to initial ROS, with significant changes in apoptosis, autophagy, and cell cycle arrest in G0/G1 or G2/M. When the dose of Chloramphenicol was increased to 5.37 mM for 6.30 mM of vitamin C, prostate hyperplasia cells reacted by ROS level drastically decreased, cell cycle arrest in G2/M, active apoptosis, and autophagy. The pro-oxidant action of 1.51 mM Erythromycin dose in prostate hyperplasia cell cultures induces changes in the apoptosis mechanisms and cell cycle arrest in G0/G1. Addition of 6.30 mM vitamin C to 1.51 mM Erythromycin dose in hyperplasia cell cultures, the pro-oxidant status determines diminished caspase 3/7 mechanism activation, but ROS level presents similar changes as Chloramphenicol dose and cell cycle arrest in G2/M. Flow cytometric analysis of cell death, oxidative stress, and cell cycle are recommended as laboratory techniques in therapeutic and diagnostic fields.
Apoptosis–Cell Cycle–Autophagy Molecular Mechanisms Network in Heterogeneous Aggressive Phenotype Prostate Hyperplasia Primary Cell Cultures Have a Prognostic Role
Elena Matei, Manuela Enciu, Mihai Cătălin Roșu, Felix Voinea, Anca Florentina Mitroi, et al.
International Journal of Molecular Sciences, 2024
Our study highlights the apoptosis, cell cycle, DNA ploidy, and autophagy molecular mechanisms network to identify prostate pathogenesis and its prognostic role. Caspase 3/7 expressions, cell cycle, adhesion glycoproteins, autophagy, nuclear shrinkage, and oxidative stress by flow-cytometry analysis are used to study the BPH microenvironment’s heterogeneity. A high late apoptosis expression by caspases 3/7 activity represents an unfavorable prognostic biomarker, a dependent predictor factor for cell adhesion, growth inhibition by arrest in the G2/M phase, and oxidative stress processes network. The heterogeneous aggressive phenotype prostate adenoma primary cell cultures present a high S-phase category (>12%), with an increased risk of death or recurrence due to aneuploid status presence, representing an unfavorable prognostic biomarker, a dependent predictor factor for caspase 3/7 activity (late apoptosis and necrosis), and cell growth inhibition (G2/M arrest)-linked mechanisms. Increased integrin levels in heterogenous BPH cultures suggest epithelial–mesenchymal transition (EMT) that maintains an aggressive phenotype by escaping cell apoptosis, leading to the cell proliferation necessary in prostate cancer (PCa) development. As predictor biomarkers, the biological mechanisms network involved in apoptosis, the cell cycle, and autophagy help to establish patient prognostic survival or target cancer therapy development.
Same Organ, Two Cancers: Complete Analysis of Renal Cell Carcinomas and Upper Tract Urothelial Carcinomas
Sorin Vamesu, Oana Andreea Ursica, Serban Eduard Milea, Mariana Deacu, Mariana Aschie, et al.
Medicina Lithuania, 2024
Background and Objectives: Renal cell carcinomas and upper tract urothelial carcinomas are types of malignancies that originate in the kidneys. Each of these examples shows an increasing trend in the frequency and the mortality rate. This study aims to comprehensively define carcinomas by analyzing clinical, paraclinical, and histological aspects to predict aggressiveness and mortality. Materials and Methods: We conducted a retrospective investigation on a group of patients suspected of kidney cancers. Results: We identified 188 cases. We observed a higher mortality rate and older age in individuals with urothelial carcinomas. Anemia, acute kidney injury, hematuria, and perineural invasion were the main risk factors that predicted their mortality. Tumor size in renal cell carcinomas correlates with the presence of necrosis and sarcomatoid areas. Factors that indicate a higher rate of death are older age, exceeding the renal capsule, a lesion that includes the entire kidney, lymphovascular invasion, acute kidney injury, and anemia. Conclusions: Even if they originate at the renal level, and the clinical–paraclinical picture is similar, the histopathological characteristics make the difference. In addition, to these are added the previously mentioned common parameters that can represent important prognostic factors. In conclusion, the characteristics commonly identified in one type of cancer may act as risk factors for the other tumor. The detected data include threshold values and risk factors, making a significant contribution to the existing literature.
The impact of MYD88 and PIM1 in mature large B-cell non-Hodgkin lymphomas: Defining element of their evolution and prognosis
Miruna Cristian, Mariana Așchie, Anca-Florentina Mitroi, Mariana Deacu, Mădălina Boșoteanu, et al.
Medicine United States, 2024
CMV and HIV Coinfection in Women from a Region in Eastern Europe
Stela Halichidis, Mariana Aschie, Georgeta Camelia Cozaru, Mihaela Manea, Nicolae Dobrin, et al.
Journal of Personalized Medicine, 2023
A retrospective study of nonneoplastic and neoplastic disorders of the salivary glands
Sorin Vamesu, Oana Andreea Ursica, Ana Maria Gurita, Raluca Ioana Voda, Mariana Deacu, et al.
Medicine United States, 2023
NET G3 vs NEC: p53 and Rb1 Immunolabeling in High-grade Gastrointestinal Neuroendocrine Neoplasms-Is It Enough for the Differential Diagnosis?
Alexandra Dinu, Mariana Aschie, Georgeta Camelia Cozaru, Anca Florentina Mitroi, Catalin Nicolae Grasa, et al.
Journal of Gastrointestinal and Liver Diseases, 2023
The Role of Pathogenesis Associated with the Tumor Microclimate in the Differential Diagnosis of Uterine Myocytic Tumors
Madalina Bosoteanu, Mariana Deacu, Mariana Aschie, Sorin Vamesu, Georgeta Camelia Cozaru, et al.
Journal of Clinical Medicine, 2023
Tissue and Circulating MicroRNA-31, MicroRNA-200b, and MicroRNA-200c Reflects Disease Activity in Crohn‘s Disease Patients: Results from the BIOMIR Study
Cristina Tocia, Andrei Dumitru, Bogdan Mateescu, Lucian Negreanu, Monica State, et al.
Journal of Gastrointestinal and Liver Diseases, 2023
Diffuse large B cell lymphoma CD5-positive arising in an immune deficiency and immune dysregulation setting: A case report and brief review of the literature
Miruna Cristian, Radu Andrei Baz, Andreea Georgiana Stoica, Mariana Așchie, Maria Mihaela Ghinea, et al.
Medicine United States, 2023
TCF7L2, CASC8, and GREM1 polymorphism and colorectal cancer in south-eastern Romanian population
Anca Florentina Mitroi, Nicoleta Leopa, Eugen Dumitru, Andrei Dumitru, Cristina Tocia, et al.
Medicine United States, 2023
Implications of Cellular Immaturity in Necrosis and Microvascularization in Glioblastomas IDH-Wild-Type
Cristian Ionut Orasanu, Mariana Aschie, Mariana Deacu, Madalina Bosoteanu, Sorin Vamesu, et al.
Clinics and Practice, 2022
Proteomics and genomics of a monomorphic epitheliotropic intestinal T-cell lymphoma: An extremely rare case report and short review of literature
Mădălina Boșoteanu, Miruna Cristian, Mariana Așchie, Mariana Deacu, Anca Florentina Mitroi, et al.
Medicine United States, 2022
KRAS, NRAS, BRAF, PIK3CA, and AKT1 signatures in colorectal cancer patients in south-eastern Romania
Costel Stelian Brinzan, Mariana Aschie, Georgeta Camelia Cozaru, Mariana Deacu, Eugen Dumitru, et al.
Medicine United States, 2022
Association of TCF7L2, CASC8 and GREM1 Polymorphisms in Patients with Colorectal Cancer and Type II Diabetes Mellitus
Anca Florentina Mitroi, Nicoleta Leopa, Eugen Dumitru, Costel Brînzan, Cristina Tocia, et al.
Genes, 2022
Characterization of the Tumor Microenvironment and the Biological Processes with a Role in Prostatic Tumorigenesis
Cristina-Anita Ionescu, Mariana Aschie, Elena Matei, Georgeta Camelia Cozaru, Mariana Deacu, et al.
Biomedicines, 2022
Changes in Platelet Function in Preterm Newborns with Prematurity Related Morbidities
Irina Franciuc, Elena Matei, Mariana Aschie, Anca Mitroi, Anca Chisoi, et al.
Children, 2022
HPV and HIV Coinfection in Women from a Southeast Region of Romania—PICOPIV Study
Simona Claudia Cambrea, Mariana Aschie, Ghiulendan Resul, Anca Florentina Mitroi, Anca Chisoi, et al.
Medicina Lithuania, 2022
Dinosaur Tail Appendix in Trisomy 13
Mădălina Boșoteanu, Cristian Ionuț Orășanu, Mariana Așchie, Mariana Deacu, Georgeta Camelia Cozaru, et al.
Fetal and Pediatric Pathology, 2022
Colorectal cancer in patients with diabetes mellitus
Romanian Journal of Diabetes Nutrition and Metabolic Diseases, 2021
Molecular profiling of the colon cancer in South-Eastern Romania: Results from the MERCUR study
Razvan Catalin Popescu, Cristina Tocia, Costel Brînzan, Georgeta Camelia Cozaru, Mariana Deacu, et al.
Medicine United States, 2021
Biomarkers involved in evaluation of platelets function in South-Eastern Romanian patients with hematological malignancies subtypes
Elena Matei, Mariana Aschie, Anca Florentina Mitroi, Mihaela Maria Ghinea, Emma Gheorghe, et al.
Medicine United States, 2021
Assessment of programmed death-ligand 1 receptor immunohistochemical expression and its association with tumor-infiltrating lymphocytes and p53 status in triple-negative breast cancer
Mariana Deacu, Liliana-Ana Tuţă, Mădălina Boşoteanu, Mariana Aşchie, Anca Florentina Mitroi, et al.
Romanian Journal of Morphology and Embryology, 2021
The diagnostic value of miR-92a, -143, and -145 expression levels in patients with colorectal adenocarcinoma from Romania
Costel Brînzan, Mariana Aşchie, Georgeta Cozaru, Eugen Dumitru, Anca Mitroi
Medicine United States, 2020
Genetic antibiotic resistance of helicobacter pylori in South-Eastern Romania
Eugen Dumitru, Luana Alexandrescu, Anda Carmen Hanu, Cristina Tocia, Georgeta Camelia Cozaru, et al.
Journal of Gastrointestinal and Liver Diseases, 2020
Molecular expression profiles of selected microRNAs in colorectal adenocarcinoma in patients from south-eastern part of Romania
Costel Brînzan, Mariana Aşchie, Elena Matei, Anca Mitroi, Georgeta Cozaru
Medicine United States, 2019
The mutation profiles of KRAS and BRAF genes in a Romanian colorectal cancer cohort
Costel Brinzan, Mariana Aschie, Catalin Nicolae Grasa, Anca Florentina Mitroi, Elena Matei, et al.
Revista De Chimie, 2019
The genetic profile of thrombophilia in reproductive disorders
Georgeta Camelia Cozaru, Mariana Aschie, Anca Mitroi, Costel Brinzan, Anca Chisoi
Revista De Chimie, 2019
Synchronous association of two types of indolent lymphomas
Mariana Aschie, Andreea Georgiana Stoica, Anca Florentina Mitroi, Georgeta Camelia Cozaru, Gabriela Stanciu, et al.
Revista De Chimie, 2018
A boy with 13.34-Mb interstitial deletion of chromosome 4p15: A new case report and review of the literature
Anca Florentina Mitroi, Mariana Aschie, Adriana Apostol, Costel Brinzan, Georgeta Cozaru, et al.
Medicine United States, 2017
ETHICAL AND GENETIC ASPECTS REGARDING PRESYMPTOMATIC TESTING FOR NEURODEGENERATIVE DISEASES
Revista Medico Chirurgicala A Societatii De Medici Si Naturalisti Din Iasi, 2016
Relationship between imunohistochemical markers and chromosomal abnormalities in nodal diffuse large B-cell lymphoma
Acta Medica Mediterranea, 2015
Clinico-pathological and molecular subtypes of male breast carcinoma according to immunohistochemistry
Romanian Journal of Morphology and Embryology, 2013
Classification of breast carcinomas according to gene expression profiles.
Journal of Medicine and Life, 2013
Glucocorticoids effect on bone mineral density in menopausal patients with rheumatoid arthritis
Archives of the Balkan Medical Union, 2012
Balanced chromosomal rearrangements in couples with spontaneous recurrent abortions
Archives of the Balkan Medical Union, 2012

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